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Prof Stephanie Dakin

BVetMed PhD FHEA FRCVS
Stephanie Dakin
  • Location: Oxfordshire
  • Year of Fellowship: 2025
  • Route to Fellowship: Meritorious Contributions to Knowledge

Field of work

Universities and colleges

Areas of special interest

  • Musculoskeletal Sciences
  • Inflammation
  • Fibrosis

Areas of support

  • Collaborative research
  • One Health Agenda
  • Professional mentoring

Professional positions

  • Professor of Musculoskeletal Sciences, Nuffield Department of Orthopaedics, Rheumatology and Musculoskeletal Sciences, Senior Research Fellow, Green Templeton College, University of Oxford

Awards

  • Fellow of the Higher Education Academy
  • PG Cert Teaching, Learning & Higher Education
  • Novartis Institute for Global Research Global Scholar
  • Excellent Teacher Award, Medical Sciences Division, University of Oxford
  • 100 Women of Oxford Medical Sciences

Biography

Stephanie graduated as a veterinary surgeon from Royal Veterinary College in 2003. After undertaking an internship specialising in equine orthopaedics at the Animal Health Trust, she then spent 10 years in equine practice across the UK. In 2012, she successfully completed a PhD on the role of inflammation in equine tendinopathy at the Royal Veterinary College. Stephanie then moved to the Nuffield Department of Orthopaedics, Rheumatology & Musculoskeletal Sciences, Oxford University to advance and translate her research from horses to humans and was awarded consecutive Research Fellowships from Versus Arthritis (Foundation Fellowship), an Oxford-UCB Prize Fellowship in Biomedical Sciences and subsequently a Versus Arthritis Career Development Fellowship.
Stephanie is currently Full Professor of Musculoskeletal Sciences and a Senior Research Fellow at Green Templeton College at Oxford University. She is Principal Investigator of the Dakin Laboratory Group, which undertakes translational research to identify the cellular and molecular basis underpinning soft tissue joint disease affecting horses and humans. Understanding the cellular and molecular basis driving chronic inflammation and fibrosis will inform new therapeutic approaches to treat these common and disabling musculoskeletal injuries affecting both species.

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